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This deck focuses on Head Spine And Neurotrauma, giving you a quick way to review the definitions, rules, and examples that matter most for NREMT Paramedic Level.
Study Head Spine And Neurotrauma in NREMT Paramedic Level with focused flashcards that help you recognize the idea, recall the key rule, and apply it in practice-style prompts.
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What is the key airway contraindication in suspected basilar skull fracture?
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Avoid nasopharyngeal airway (and nasal intubation). Nasal routes risk intracranial penetration through cribriform plate fractures in basilar skull injuries.
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This deck focuses on Head Spine And Neurotrauma, giving you a quick way to review the definitions, rules, and examples that matter most for NREMT Paramedic Level.
Work through these flashcards in short sessions. Try to answer each prompt before flipping the card, then revisit any cards you miss until the explanation feels automatic.
Answer: Avoid nasopharyngeal airway (and nasal intubation). Nasal routes risk intracranial penetration through cribriform plate fractures in basilar skull injuries.
Answer: Hypertension, bradycardia, irregular respirations. These vital sign changes result from brainstem compression and compensatory responses to elevated intracranial pressure.
Answer: Head-of-bed elevation about 30∘ with neutral alignment. This positioning facilitates cerebral venous drainage, reducing intracranial pressure without compromising spinal alignment.
Answer: Decorticate posturing (flexor). Flexor posturing indicates cortical or subcortical damage above the brainstem, often allowing better functional recovery.
Answer: Cover with moist sterile dressing; do not apply pressure. Moist dressings prevent desiccation of exposed tissue, while avoiding pressure minimizes further herniation or contamination.
Answer: Clear otorrhea or rhinorrhea consistent with CSF leak. Clear fluid drainage from ears or nose post-trauma indicates dural tear allowing CSF escape, confirmed by beta-2 transferrin testing.
Answer: GCS ≤8. Severe TBI is classified by GCS scores indicating profound neurological impairment requiring immediate intervention.
Answer: Brown-Séquard syndrome (hemisection). Hemisection interrupts ipsilateral corticospinal and dorsal column tracts while sparing contralateral spinothalamic tracts.
Answer: Transient loss of reflexes and flaccid paralysis below injury. Spinal shock involves temporary areflexia and paralysis due to disrupted descending neural pathways immediately after injury.
Answer: Manual in-line stabilization (MILS). MILS minimizes cervical spine movement during airway management to prevent exacerbation of potential unstable injuries.
Answer: Neurogenic: hypotension with bradycardia and warm skin. Neurogenic shock from sympathetic disruption causes vasodilation and bradycardia, unlike tachycardia and vasoconstriction in hypovolemic states.
Answer: Neurogenic shock with hypotension and bradycardia. High cervical lesions disrupt sympathetic innervation, leading to unopposed parasympathetic effects during positive pressure ventilation.
Answer: Subdural hematoma. Venous bleeding from torn bridging veins accumulates slowly, with higher incidence in elderly due to brain atrophy and anticoagulation.
Answer: Administer benzodiazepine per protocol and protect airway. Benzodiazepines terminate seizures to prevent increased metabolic demand and ICP elevation, with airway protection against aspiration.
Answer: EtCO2 35 to 45 mmHg. Normocapnia prevents vasoconstriction or vasodilation that could alter cerebral blood flow and intracranial pressure.
Answer: Decerebrate posturing (extensor). Extensor posturing reflects dysfunction at the midbrain or pons level, correlating with poorer recovery outcomes.
Answer: Sedative per protocol (for example, ketamine or benzodiazepine). Sedation controls agitation that could raise intracranial pressure, ensuring patient and provider safety during transport.
Answer: Maintain SpO2 ≥94% (avoid hypoxia). Hypoxia worsens cerebral ischemia, so targeting this level ensures sufficient oxygen delivery to injured brain tissue.
Answer: Prevent secondary brain injury (avoid hypoxia and hypotension). Secondary injuries exacerbate primary brain damage, so prehospital care prioritizes oxygenation and blood pressure maintenance to optimize cerebral perfusion.
Answer: EtCO2 30 to 35 mmHg (short-term only). Temporary hypocapnia induces cerebral vasoconstriction to reduce intracranial pressure during acute herniation signs.
Answer: Unilateral fixed, dilated pupil. This finding reflects oculomotor nerve compression from temporal lobe herniation through the tentorial notch.
Answer: Central cord syndrome. Hyperextension damages central cervical tracts, disproportionately affecting upper extremities in patients with spondylosis.
Answer: SBP ≥110 mmHg. Maintaining adequate cerebral perfusion pressure reduces secondary ischemic injury and improves outcomes in TBI patients.
Answer: Both are signs of basilar skull fracture. Battle sign (postauricular ecchymosis) and raccoon eyes (periorbital ecchymosis) both signify fractures involving the skull base.
Answer: Epidural hematoma. Arterial bleeding from middle meningeal artery rupture causes rapid hematoma expansion after initial symptom resolution.