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This deck focuses on Acute Coronary Syndromes And Ischemic Syndromes, giving you a quick way to review the definitions, rules, and examples that matter most for NREMT Paramedic Level.
Study Acute Coronary Syndromes And Ischemic Syndromes in NREMT Paramedic Level with focused flashcards that help you recognize the idea, recall the key rule, and apply it in practice-style prompts.
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Which blood pressure threshold is a common contraindication to nitroglycerin in ACS?
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Systolic blood pressure <90 mmHg (or signs of shock). Low SBP indicates hemodynamic instability where nitroglycerin's vasodilatory effects could worsen hypotension and perfusion.
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This deck focuses on Acute Coronary Syndromes And Ischemic Syndromes, giving you a quick way to review the definitions, rules, and examples that matter most for NREMT Paramedic Level.
Work through these flashcards in short sessions. Try to answer each prompt before flipping the card, then revisit any cards you miss until the explanation feels automatic.
Answer: Systolic blood pressure <90 mmHg (or signs of shock). Low SBP indicates hemodynamic instability where nitroglycerin's vasodilatory effects could worsen hypotension and perfusion.
Answer: Fentanyl (titrate to effect per protocol). Opioids like fentanyl reduce pain and sympathetic drive, decreasing myocardial oxygen demand in persistent ACS symptoms.
Answer: Give IV fluid bolus; suspect right ventricular infarction. Hypotension post-nitroglycerin in inferior STEMI suggests RV involvement, where fluids restore preload without overloading the left ventricle.
Answer: Hypotension with pulmonary edema and signs of poor perfusion. This presentation indicates pump failure from extensive myocardial damage, leading to low output and congestion in ACS.
Answer: Anterior (anteroseptal) myocardial infarction. Precordial leads V1-V4 correspond to the anterior septum and wall, supplied by the left anterior descending artery, indicating occlusion there.
Answer: Right ventricular infarction (often with inferior STEMI). RV infarction relies on preload for output, so nitroglycerin's venodilation can precipitate hypotension and shock.
Answer: Aspirin 162–324 mg, chewable. Aspirin inhibits platelet aggregation via cyclooxygenase blockade, reducing thrombus progression in ACS when given early.
Answer: NSTEMI has positive cardiac biomarkers; unstable angina does not. Cardiac biomarkers like troponin indicate myocardial cell death in NSTEMI, distinguishing it from unstable angina where ischemia occurs without necrosis.
Answer: Activate rapid reperfusion pathway and transport to PCI-capable facility. Early reperfusion via PCI minimizes myocardial damage, so prompt activation and transport optimize outcomes in STEMI.
Answer: Inferior wall myocardial infarction. Leads II, III, and aVF view the inferior cardiac wall, so elevation there localizes ischemia to the right coronary artery territory.
Answer: ST depression and/or T-wave inversion. These changes indicate subendocardial ischemia due to reduced coronary perfusion without complete occlusion or transmural involvement.
Answer: Oxygen to maintain 94\% SpO2. Supplemental oxygen corrects hypoxemia to ensure adequate myocardial oxygenation, targeting SpO2 ≥94% to avoid hyperoxia risks.
Answer: ST-segment elevation in a contiguous lead set. ST elevation reflects acute transmural ischemia or infarction from epicardial coronary occlusion, distinguishing it from subendocardial processes.
Answer: Do not administer routine oxygen; monitor and reassess. Routine oxygen in normoxic patients lacks benefit and may cause vasoconstriction, so monitoring suffices per evidence-based guidelines.
Answer: Lead V4R (right-sided ECG). V4R provides a right-sided view to detect ST elevation indicative of right ventricular involvement, often from proximal right coronary occlusion.
Answer: Acute thrombotic occlusion after plaque rupture. Plaque rupture exposes thrombogenic material, promoting platelet aggregation and thrombus formation that fully occludes the coronary artery in STEMI.
Answer: PDE-5 inhibitors taken recently (for example, sildenafil). PDE-5 inhibitors potentiate nitroglycerin's nitric oxide-mediated vasodilation, risking severe hypotension or cardiovascular collapse.
Answer: Bradycardia or AV block (especially 2nd-degree type I). Inferior MI often involves the right coronary artery supplying the AV node, causing ischemic conduction delays like bradycardia or blocks.
Answer: ST-elevation myocardial infarction (STEMI). STEMI involves complete coronary occlusion leading to full-thickness myocardial necrosis, evidenced by persistent ST-segment elevation on ECG and elevated cardiac biomarkers.
Answer: Nitroglycerin 0.4 mg SL every 5 minutes as tolerated. Nitroglycerin relieves ischemia by vasodilating coronary arteries and reducing preload, repeated as needed if hemodynamically stable.
Answer: At least 2 contiguous leads. Diagnostic criteria require ST elevation in at least two anatomically contiguous leads to confirm regional myocardial involvement in STEMI.
Answer: Hypotension, clear lungs, and jugular venous distension. This triad reflects right ventricular dysfunction causing reduced preload and cardiac output without left-sided failure or congestion.
Answer: Non–ST-elevation myocardial infarction (NSTEMI). NSTEMI results from partial coronary occlusion causing subendocardial necrosis, detected by elevated cardiac biomarkers without ST elevation on ECG.
Answer: Lateral wall myocardial infarction. These leads reflect the lateral wall, perfused by the left circumflex artery, localizing the infarction to that region.