Fluid And Electrolyte Imbalances
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NCLEX-RN › Fluid And Electrolyte Imbalances
A 70-year-old client with heart failure is admitted for fluid volume excess and is receiving a loop diuretic. The client has bilateral crackles and 3+ pitting edema. Vital signs: blood pressure 156/88 mm Hg, heart rate 98/min, respiratory rate 24/min, oxygen saturation 92% on 2 L/min nasal cannula. Labs: sodium 126 mEq/L (low), potassium 4.1 mEq/L. Which symptom indicates a need for FURTHER assessment?
New onset confusion and headache
Nocturia occurring twice nightly
Mild exertional dyspnea that resolves with rest
Bilateral ankle edema that improves with leg elevation
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances, specifically recognizing cerebral symptoms of severe hyponatremia. The key assessment finding is new onset confusion and headache in a patient with hyponatremia (126 mEq/L) and fluid overload. New onset confusion and headache (A) is the BEST choice for further assessment because these are signs of cerebral edema from severe hyponatremia requiring immediate intervention. Bilateral ankle edema (B), mild exertional dyspnea (C), and nocturia (D) are expected findings in heart failure with fluid overload and don't indicate acute deterioration. The decision-making principle is that neurological symptoms in hyponatremia indicate cerebral edema requiring urgent treatment to prevent seizures or coma. A transferable strategy is to prioritize neurological assessment in hyponatremic patients, as mental status changes herald life-threatening complications.
A 6-year-old child with acute gastroenteritis has had vomiting and watery diarrhea for 2 days and is lethargic with decreased tears. Vital signs: heart rate 132/min, respiratory rate 24/min, blood pressure 88/50 mm Hg; capillary refill 4 seconds. Labs: potassium 2.9 mEq/L (low), bicarbonate 18 mEq/L (low). What is the nurse's PRIORITY action for this child?
Initiate isotonic intravenous fluids and anticipate potassium replacement as ordered
Administer an antidiarrheal medication as prescribed to reduce stool output
Offer clear liquids only and reassess hydration status in 2 hours
Restrict fluids to prevent worsening electrolyte dilution
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances, specifically hypovolemic shock with hypokalemia in a pediatric patient. The key assessment finding is severe dehydration with hypotension, tachycardia, delayed capillary refill, and critical hypokalemia (2.9 mEq/L). Initiating isotonic IV fluids and anticipating potassium replacement (B) is the BEST choice because it addresses the immediate circulatory collapse while preparing for electrolyte correction once urine output is established. Antidiarrheal medication (A) doesn't address the life-threatening hypovolemia; clear liquids only (C) is insufficient for severe dehydration with shock; fluid restriction (D) would be fatal in hypovolemic shock. The decision-making principle is that pediatric hypovolemic shock requires rapid isotonic fluid resuscitation, with potassium replacement after establishing adequate perfusion and urine output. A transferable strategy is to prioritize circulatory stabilization in pediatric dehydration before correcting electrolyte imbalances, as potassium replacement requires functioning kidneys.
A 6-year-old child with gastroenteritis has diarrhea and abdominal cramping. Assessment: muscle weakness; vital signs: heart rate 124/min, blood pressure 92/56 mm Hg. Labs: potassium 3.1 mEq/L (3.5–5.0). What is the nurse's PRIORITY action?
Administer intravenous calcium gluconate to prevent dysrhythmias
Obtain a stool culture before initiating any treatment
Encourage a low-potassium diet to prevent hyperkalemia
Administer potassium replacement as prescribed after verifying adequate urine output and continue hydration therapy
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The priority concern is hypokalemia with muscle weakness and tachycardia in a child with gastroenteritis. Administering potassium replacement after verifying urine output and continuing hydration is the best choice to correct safely. Calcium gluconate is for hyperkalemia; low-potassium diet is counterproductive; stool culture delays treatment. The decision-making principle is to hydrate first then replace potassium in pediatric GI losses. Verify renal function. A transferable strategy is to integrate hydration and electrolyte correction in children with diarrhea and cramps.
An 82-year-old client with a history of dementia and chronic kidney disease stage 3 is brought to the emergency department from a nursing facility for acute confusion and lethargy after 2 days of poor oral intake. Assessment shows dry mucous membranes, poor skin turgor, and urine output 15 mL/hr; vital signs: blood pressure 92/54 mm Hg, heart rate 112/min, respiratory rate 18/min, temperature 37.1°C (98.8°F). Labs: sodium 156 mEq/L (135–145), blood urea nitrogen 38 mg/dL (7–20), creatinine 1.8 mg/dL (0.6–1.3). What is the nurse's PRIORITY action for this client?
Encourage oral free-water intake and recheck sodium level in 4 hours
Administer furosemide intravenously to promote sodium excretion
Initiate seizure precautions and prepare to administer hypertonic saline as ordered
Start an intravenous infusion of 0.9% normal saline and monitor intake and output closely
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The key assessment finding is hypernatremia with signs of hypovolemia, including low blood pressure, tachycardia, dry mucous membranes, and elevated BUN and creatinine. Starting an intravenous infusion of 0.9% normal saline is the best choice because it addresses hypovolemia safely without rapidly altering sodium levels in a client with chronic kidney disease. Initiating seizure precautions and hypertonic saline is incorrect as it would worsen hypernatremia; encouraging oral free-water intake overlooks hypovolemia and poor intake history; administering furosemide would exacerbate dehydration and is not indicated. The decision-making principle is to first restore intravascular volume with isotonic fluids in hypovolemic hypernatremia before correcting sodium gradually. This prevents complications like cerebral edema from overly rapid correction. A transferable strategy is to prioritize hemodynamic stabilization in dehydrated clients with electrolyte imbalances before addressing specific lab abnormalities.
A 33-year-old client with suspected diabetes insipidus reports urinating 'every 30 minutes' and drinking large amounts of water. Vital signs: blood pressure 98/60 mm Hg, heart rate 116/min; urine output 350 mL/hr for 3 hours. Labs: sodium 154 mEq/L (135–145), urine specific gravity 1.003 (1.005–1.030). What is the nurse's PRIORITY action for this client?
Begin strict intake and output measurement and notify the provider of suspected diabetes insipidus findings
Prepare to administer potassium chloride for suspected hypokalemia
Restrict all fluids to prevent further sodium elevation
Administer oral sodium tablets to correct the sodium level
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The priority concern is hypernatremia with high urine output and low specific gravity, suggestive of diabetes insipidus. Beginning strict intake and output measurement and notifying the provider is the best choice to confirm diagnosis and initiate treatment like desmopressin. Restricting fluids would worsen dehydration; oral sodium tablets are contraindicated; potassium administration is not indicated without labs. The decision-making principle is to monitor fluid balance precisely in suspected diabetes insipidus to guide hormone replacement. Avoid rapid sodium correction to prevent complications. A transferable strategy is to use I&O trends to identify endocrine-related fluid imbalances early in polyuric clients.
A 3-year-old child is seen in the emergency department for 2 days of vomiting and watery diarrhea. Assessment: lethargy, sunken eyes, dry mucous membranes, capillary refill 4 seconds; vital signs: heart rate 148/min, respiratory rate 28/min, blood pressure 86/50 mm Hg, temperature 37.4°C (99.3°F). Labs: potassium 3.0 mEq/L (3.5–5.0), bicarbonate 18 mEq/L (22–28). What is the nurse's PRIORITY action for this child?
Administer a rapid isotonic intravenous fluid bolus per pediatric protocol and reassess perfusion
Prepare to administer insulin with dextrose to shift potassium into cells
Start oral potassium supplements before fluid replacement
Restrict fluids to prevent worsening diarrhea
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The priority concern is hypovolemic shock from dehydration, evidenced by tachycardia, hypotension, prolonged capillary refill, and lethargy. Administering a rapid isotonic intravenous fluid bolus is the best choice to restore perfusion and prevent organ damage in pediatric dehydration. Starting oral potassium delays volume restoration; restricting fluids worsens dehydration; insulin with dextrose is for hyperkalemia, not applicable here. The decision-making principle is to prioritize fluid resuscitation in dehydrated children before addressing electrolyte deficits like hypokalemia. Ongoing monitoring of vital signs and urine output guides further therapy. A transferable strategy is to assess hydration status using clinical signs like capillary refill and administer boluses iteratively in pediatric gastroenteritis cases.
A 69-year-old client with heart failure reports worsening orthopnea and has gained 2.7 kg (6 lb) in 5 days. Assessment: crackles throughout, 3+ edema; vital signs: blood pressure 172/94 mm Hg, heart rate 110/min, respiratory rate 28/min, oxygen saturation 88% on room air. Labs: sodium 124 mEq/L (135–145). What is the nurse's PRIORITY action?
Start seizure precautions and administer hypotonic intravenous fluids
Collect a urine specimen for sodium level before intervening
Encourage increased free-water intake to correct sodium level
Apply supplemental oxygen and position the client upright to improve ventilation
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The priority concern is acute respiratory distress with low oxygen saturation and tachypnea in heart failure with hyponatremia. Applying supplemental oxygen and positioning upright is the best choice to improve ventilation immediately. Seizure precautions and hypotonic fluids address hyponatremia but not urgently; increasing water worsens overload; urine collection delays care. The decision-making principle is to prioritize oxygenation in fluid overload states before correcting electrolytes. Follow with diuretics and restriction. A transferable strategy is to apply ABC priorities in heart failure exacerbations with concurrent hyponatremia.
A 49-year-old client is 8 hours post-op after cholecystectomy and has had repeated vomiting. Assessment: weakness and paresthesias; vital signs: blood pressure 126/74 mm Hg, heart rate 62/min. Labs: potassium 3.2 mEq/L (3.5–5.0). Which laboratory value change requires IMMEDIATE intervention by the nurse?
Glucose increases from 96 to 110 mg/dL
Chloride increases from 101 to 103 mEq/L
Sodium increases from 138 to 140 mEq/L
Potassium decreases from 3.6 to 3.2 mEq/L
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The key assessment finding is hypokalemia with weakness and paresthesias post-operatively from vomiting. The potassium decrease from 3.6 to 3.2 mEq/L requires immediate intervention to prevent cardiac or neuromuscular complications. Minor increases in sodium, chloride, and glucose are not critical. The decision-making principle is to address dropping potassium promptly in clients with GI losses. Ensure safe replacement. A transferable strategy is to monitor labs serially after surgery and intervene on downward trends in electrolytes.
A 74-year-old client with heart failure is receiving intravenous furosemide and now reports dizziness and headache. Assessment: crackles improved, but client appears drowsy; vital signs: blood pressure 136/78 mm Hg, heart rate 88/min. Labs: sodium 122 mEq/L (135–145). Which laboratory value change requires IMMEDIATE intervention by the nurse?
Chloride decreases from 102 to 100 mEq/L
Sodium decreases from 130 to 122 mEq/L
Blood urea nitrogen decreases from 24 to 18 mg/dL
Creatinine decreases from 1.2 to 1.0 mg/dL
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The key assessment finding is hyponatremia with drowsiness and headache in a heart failure client receiving diuretics. The sodium decrease from 130 to 122 mEq/L requires immediate intervention to prevent seizures or coma. Decreases in BUN, chloride, and creatinine are not critical and may reflect dilution or improved renal function. The decision-making principle is to monitor for rapid sodium drops during diuresis and adjust therapy to avoid severe hyponatremia. Fluid restriction and hypertonic saline may be needed cautiously. A transferable strategy is to track serial electrolytes during diuretic therapy in heart failure to intervene on worsening imbalances promptly.
A 31-year-old client with known diabetes insipidus is admitted for evaluation after stopping prescribed medication. Assessment: dry mucous membranes, tachycardia; urine output 4.5 L over 12 hours. Labs: sodium 157 mEq/L (135–145). Which laboratory value change requires IMMEDIATE intervention by the nurse?
Calcium increases from 9.0 to 9.6 mg/dL
Chloride decreases from 103 to 100 mEq/L
Potassium increases from 4.0 to 4.6 mEq/L
Sodium increases from 148 to 157 mEq/L
Explanation
This question tests clinical judgment in managing fluid and electrolyte imbalances. The key assessment finding is hypernatremia with high urine output and dehydration in untreated diabetes insipidus. The sodium increase from 148 to 157 mEq/L requires immediate intervention to prevent severe neurological effects. Minor increases in potassium, decreases in chloride, and increases in calcium are not urgent. The decision-making principle is to act on rising sodium in polyuric states with fluid and hormone therapy. Monitor for rapid changes. A transferable strategy is to resume medications promptly and hydrate in clients with known endocrine fluid disorders.