What this deck covers
This deck focuses on Sepsis And Shock Recognition And Priorities, giving you a quick way to review the definitions, rules, and examples that matter most for Nclexrn.
Study Sepsis And Shock Recognition And Priorities in Nclexrn with focused flashcards that help you recognize the idea, recall the key rule, and apply it in practice-style prompts.
0% Complete
What bedside finding best indicates poor tissue perfusion in shock?
Tap card or press Space to flip
Altered mental status. It reflects cerebral hypoperfusion due to inadequate oxygen delivery, signaling systemic tissue compromise in shock states.
How well did you know it?
Card 1 / 25
Space to flip · ← / → to move · once flipped, → Got it · ← Still learning
This deck focuses on Sepsis And Shock Recognition And Priorities, giving you a quick way to review the definitions, rules, and examples that matter most for Nclexrn.
Work through these flashcards in short sessions. Try to answer each prompt before flipping the card, then revisit any cards you miss until the explanation feels automatic.
Answer: Altered mental status. It reflects cerebral hypoperfusion due to inadequate oxygen delivery, signaling systemic tissue compromise in shock states.
Answer: Large-volume rapid fluid bolus. Excessive fluids exacerbate pulmonary congestion and impair cardiac output in volume-overloaded cardiogenic shock.
Answer: Target MAP = 65 mm Hg. This threshold ensures adequate organ perfusion, as lower values increase risk of organ failure in shock resuscitation.
Answer: Distributive shock (septic shock). Vasodilation in distributive shock causes hyperdynamic circulation, leading to these early clinical manifestations before decompensation.
Answer: Serum lactate. Elevated levels indicate anaerobic metabolism from tissue hypoperfusion, guiding resuscitation and prognosis in sepsis.
Answer: Control bleeding and give rapid isotonic fluids and blood products as indicated. Stopping hemorrhage and restoring volume are essential to prevent further hypoperfusion and organ damage in hypovolemic shock.
Answer: Pulmonary edema with hypotension and signs of poor cardiac output. These indicate pump failure with fluid overload, distinguishing it from vasodilatory hypotension in septic shock.
Answer: Administer broad-spectrum antibiotics within 1 hour of recognition. Timely administration reduces mortality by rapidly targeting the infectious source in sepsis management guidelines.
Answer: Suspected infection plus fever or hypothermia, tachycardia, tachypnea, altered mentation. These signs reflect systemic inflammatory response syndrome (SIRS) criteria combined with infection, indicating potential organ dysfunction in early sepsis.
Answer: Sepsis with persistent hypotension needing vasopressors and elevated lactate despite fluids. This definition from Sepsis-3 criteria identifies severe hypoperfusion and organ dysfunction requiring aggressive intervention beyond fluids alone.
Answer: Urine output = 0.5 mL/kg/hr. This rate indicates adequate renal perfusion and glomerular filtration, serving as a key goal in monitoring shock resuscitation efficacy.
Answer: Intramuscular epinephrine. It rapidly reverses vasodilation and bronchoconstriction, stabilizing hemodynamics in anaphylaxis-induced distributive shock.
Answer: Obtain cultures first if it does not delay antibiotics; then start broad-spectrum antibiotics. Cultures preserve microbial identification for targeted therapy, but antibiotics must not be delayed to prevent progression of infection.
Answer: Lactate = 2 mmol/L is abnormal; higher levels indicate worse hypoperfusion. Levels above this threshold correlate with increased mortality and need for aggressive resuscitation in hypoperfused states.
Answer: Apply supplemental oxygen to maintain adequate oxygenation and reduce work of breathing. It supports oxygen delivery and eases respiratory effort, preventing fatigue and deterioration in septic patients.
Answer: Oxygen, IV access, lactate, cultures, fluids, then antibiotics within 1 hour. This order aligns with the Surviving Sepsis Campaign hour-1 bundle to stabilize and treat without delays in critical interventions.
Answer: Perform hand hygiene and implement appropriate isolation/standard precautions immediately. Prevents transmission of potential pathogens, protecting patients and staff in accordance with infection control standards.
Answer: Tachycardia. It represents a compensatory response to decreased stroke volume or hypovolemia, appearing before hypotension in shock progression.
Answer: Patient with suspected infection plus new confusion and hypotension. New organ dysfunction like confusion and hypotension signals severe sepsis requiring immediate intervention to prevent shock.
Answer: Assess airway and perfusion, verify infusion and IV patency, then titrate per protocol. Systematic assessment ensures patient stability and proper drug delivery before adjusting therapy to avoid errors in shock management.
Answer: Persistent hypotension and rising lactate despite fluid resuscitation. These signs indicate refractory hypoperfusion and need for vasopressors, marking the transition to septic shock per clinical definitions.
Answer: Hypovolemic shock. Volume depletion triggers compensatory vasoconstriction, resulting in these signs of reduced peripheral perfusion.
Answer: Initiate sepsis protocol and support airway, breathing, and circulation. Addresses immediate life-threatening instability by prioritizing ABCs and activating evidence-based sepsis management to improve outcomes.
Answer: Norepinephrine. It effectively increases vascular tone and maintains organ perfusion with fewer arrhythmias compared to alternatives in septic shock.
Answer: Give 30 mL/kg isotonic crystalloid rapidly. This volume restores intravascular fluid to improve perfusion in hypoperfused states per Surviving Sepsis Campaign guidelines.