In the event of hypercalcemia the body has elevated blood calcium levels. As a result, osteoclasts, which are responsible for the resorption of bone matrix and the release of calcium into the bloodstream, would experience reduced function.

Osteoblasts, in contrast, help to synthesize bone and would be stimulated by high blood calcium levels to remove calcium from the blood and sequester it in bone. Osteocytes, sometimes known as osteogenitor cells, are the progenitor cells to osteoblasts.

In the event of hypercalcemia the body has elevated blood calcium levels. As a result, osteoclasts, which are responsible for the resorption of bone matrix and the release of calcium into the bloodstream, would experience reduced function.

Osteoblasts, in contrast, help to synthesize bone and would be stimulated by high blood calcium levels to remove calcium from the blood and sequester it in bone. Osteocytes, sometimes known as osteogenitor cells, are the progenitor cells to osteoblasts.

This question requires a strong understanding of factors affecting bone resorption and bone re-formation.

Bone re-formation takes place when osteoblasts use calcium and phosphate from the blood to form bone. Calcitonin stimulates osteoblasts to form bone. Parathyroid hormone (PTH), on the other hand, stimulates bone resorption and causes osteoclasts to break down bone, causing blood calcium and phosphate levels to increase.

Of the possible answers, only increased osteoclast activity would result in higher blood calcium levels. Increased osteoblast activity, decreased parathyroid hormone, and increased calcitonin would all result in lower blood calcium levels.

The skeletal system is responsible for a variety of functions in the body. Long bones contain both red and yellow bone marrow, which are the sites for fat storage and hematopoeisis respectively. Bones are also responsible for the storage of calcium and phosphate, which can be released in order to maintain normal levels in the blood.

White blood cells are derived from stem cells in the bone marrow. B-cells continue to develop in the bone marrow, but T-cells develop in the thymus.

The skeletal system is responsible for a variety of functions in the body. Long bones contain both red and yellow bone marrow, which are the sites for fat storage and hematopoeisis respectively. Bones are also responsible for the storage of calcium and phosphate, which can be released in order to maintain normal levels in the blood.

White blood cells are derived from stem cells in the bone marrow. B-cells continue to develop in the bone marrow, but T-cells develop in the thymus.

Osteocytes are differentiated osteoblasts that have become imbedded in hydroxyapatite bone matrix. The osteocytes are found in small gaps in bone matrix called lacunae and exchange nutrients with the blood using small canals called canaliculi. Lamellae are the concentric regions of osteocytes that are arranged around the central Haversian canal. The canal houses blood vessels and nerves to nourish and stimulate the osteocytes via the canaliculi.

Osteocytes are differentiated osteoblasts that have become imbedded in hydroxyapatite bone matrix. The osteocytes are found in small gaps in bone matrix called lacunae and exchange nutrients with the blood using small canals called canaliculi. Lamellae are the concentric regions of osteocytes that are arranged around the central Haversian canal. The canal houses blood vessels and nerves to nourish and stimulate the osteocytes via the canaliculi.

This question requires a strong understanding of factors affecting bone resorption and bone re-formation.

Bone re-formation takes place when osteoblasts use calcium and phosphate from the blood to form bone. Calcitonin stimulates osteoblasts to form bone. Parathyroid hormone (PTH), on the other hand, stimulates bone resorption and causes osteoclasts to break down bone, causing blood calcium and phosphate levels to increase.

Of the possible answers, only increased osteoclast activity would result in higher blood calcium levels. Increased osteoblast activity, decreased parathyroid hormone, and increased calcitonin would all result in lower blood calcium levels.

When it comes to the strength and resilience of bone, both collagen and calcium play important roles. Collagen provides bones with great tensile strength, and calcium (usually in the form of hydroxyapatite) provides bones with great compressive strength. In the event of a calcium deficiency, weight placed on bones would cause them to bend irregularly, resulting in the characteristic bending of bones found with rickets.

A deficiency in collagen would cause damage to the bone during extension or stretching forces. Bone marrow does not play an important structural role, and is more important for bone function.

Bone matrices store these slightly soluble calcium salts so that they can be released into the bloodstream in times of low blood calcium levels. As a result, these salts are an example of how the skeletal system provides mineral storage for the body.

Movement and bone cell production are also primary functions of the skeletal system, but are not linked to calcium salts. Energy storage is linked to the fats stored in the yellow marrow of the bone.