Africa sleeping sickness, also known as African trypanosomosis, is caused by a parasite. The parasite, Trypanosoma brucei rhodesiense, is carried by the Tsetse fly. The fly bite is the mechanism for the parasite entering the body. There are two variations of sleeping sickness, East African and West African.

Africa sleeping sickness, also known as African trypanosomosis, is caused by a parasite. The parasite, Trypanosoma brucei rhodesiense, is carried by the Tsetse fly. The fly bite is the mechanism for the parasite entering the body. There are two variations of sleeping sickness, East African and West African.

Mast cells are perhaps the best-known producers of histamine. Basophils also release histamine and contribute to the inflammatory response, while platelets are also capable of releasing histamine. Neutrophils are best-known for their phagocytic properties, and do not produce histamine.

Mast cells are perhaps the best-known producers of histamine. Basophils also release histamine and contribute to the inflammatory response, while platelets are also capable of releasing histamine. Neutrophils are best-known for their phagocytic properties, and do not produce histamine.

Mast cell functions in allergic responses by release histamine and other chemicals that promote inflammation. Macrophages are part of the innate immune system and function to engulf and remove debris. Neutrophils are also part of the innate immune system and have macrophagic properties, especially when fighting bacterial infection. Natural killer cells bind to and destroy pathogens without the use of antigens.

Mast cell functions in allergic responses by release histamine and other chemicals that promote inflammation. Macrophages are part of the innate immune system and function to engulf and remove debris. Neutrophils are also part of the innate immune system and have macrophagic properties, especially when fighting bacterial infection. Natural killer cells bind to and destroy pathogens without the use of antigens.

NSAIDs inhibit COX-1, inhibiting the formation of prostanglandins.

NSAIDs inhibit COX-1, inhibiting the formation of prostanglandins.

Opportunistic microbiota can form biofilms and affect patients who are immunocompromised, suchs as someone with AIDS or a chronic illness.

Resident microbiota are always present in the body and frequently have mutualistic relationships with their human hosts. Transient microbiota are present at some times, but absent at others. Pathogens affect healthy individuals, as well as those who may be immunocompromised.

Opportunistic microbiota can form biofilms and affect patients who are immunocompromised, suchs as someone with AIDS or a chronic illness.

Resident microbiota are always present in the body and frequently have mutualistic relationships with their human hosts. Transient microbiota are present at some times, but absent at others. Pathogens affect healthy individuals, as well as those who may be immunocompromised.