Anatomy Quiz: Nutrient Homeostasis And Fed Fasted States
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Nutrient Homeostasis And Fed Fasted StatesQuestion 1 of 1

A diabetic patient accidentally takes twice their normal insulin dose. Two hours later, blood glucose drops to 45 mg/dL. Which compensatory mechanism would be activated FIRST to counteract this hypoglycemia?

Pancreatic alpha cells increase glucagon secretion to stimulate hepatic glucose production
Adrenal cortex increases cortisol release to promote gluconeogenesis
Thyroid gland increases T3/T4 secretion to enhance metabolic rate
Anterior pituitary increases growth hormone release to stimulate glucose production
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Anatomy Quiz: Nutrient Homeostasis And Fed Fasted States

Practice Nutrient Homeostasis And Fed Fasted States in Anatomy with focused quiz questions that help you check what you know, review explanations, and build confidence with test-style prompts.

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Question 1

A diabetic patient accidentally takes twice their normal insulin dose. Two hours later, blood glucose drops to 45 mg/dL. Which compensatory mechanism would be activated FIRST to counteract this hypoglycemia?

  1. Pancreatic alpha cells increase glucagon secretion to stimulate hepatic glucose production (correct answer)
  2. Adrenal cortex increases cortisol release to promote gluconeogenesis
  3. Thyroid gland increases T3/T4 secretion to enhance metabolic rate
  4. Anterior pituitary increases growth hormone release to stimulate glucose production
Explanation: Glucagon secretion from pancreatic alpha cells is the first and fastest hormonal response to hypoglycemia, typically occurring within minutes. Glucagon rapidly stimulates hepatic glycogenolysis and gluconeogenesis to restore blood glucose levels. Choice B (cortisol) is a slower response that takes hours to become effective. Choice C (thyroid hormones) affects long-term metabolic rate but doesn't provide acute glucose counter-regulation. Choice D is incorrect because while growth hormone does have counter-regulatory effects, the glucagon response is much faster and represents the primary first-line defense against hypoglycemia.