Certified EKG Technician (CET) • EKG Acquisition

Stress Test Monitoring — Monitor patient condition during stress testing

Ensuring patient safety through continuous physiological surveillance during exercise and pharmacological cardiac stress testing.

A heart scan with an electrical waveform and pulsing heart.
Inside this lesson 10 sections 5 questions 9 figures

SECTION 1

Historical Context & Motivation

The concept of evaluating cardiac function under physiological stress has roots stretching back to the early twentieth century, when clinicians first recognized that a resting electrocardiogram often fails to reveal underlying coronary artery disease. At rest, the myocardium may receive adequate perfusion even through partially occluded vessels, masking ischemia that becomes clinically significant only when metabolic demand increases. This insight drove the development of cardiac stress testing — a controlled procedure in which a patient exercises on a treadmill or cycle ergometer (or receives a pharmacological agent) while continuous EKG monitoring, blood pressure measurement, and symptom assessment are performed. The role of the EKG technician during this procedure is not merely to acquire tracings; it is to serve as the first line of defense for patient safety, continuously monitoring for rhythm disturbances, ST-segment changes, hemodynamic instability, and clinical symptoms that may warrant immediate test termination.

1929 • Master Two-Step Test

Arthur Master introduced a standardized step test to provoke cardiac symptoms and record post-exercise EKG changes, establishing the earliest formalized exercise stress protocol.

1956 • Treadmill Protocols Emerge

Robert Bruce developed the Bruce treadmill protocol with graded stages of increasing speed and incline, enabling reproducible, quantifiable workload assessment that remains widely used today.

1976 • Pharmacological Stress Agents

Introduction of dipyridamole and later adenosine and dobutamine allowed stress testing for patients unable to exercise, broadening the clinical applicability of the procedure.

1997 • ACC/AHA Practice Guidelines

The American College of Cardiology and American Heart Association published comprehensive guidelines codifying indications, contraindications, monitoring parameters, and termination criteria for exercise stress testing.

2010s • Digital Continuous Monitoring

Advances in digital EKG systems enabled real-time, high-resolution 12-lead continuous monitoring with automated ST-segment trend analysis, enhancing the technician's ability to detect ischemic changes instantly.

The historical trajectory of stress testing reveals a central tension: how can clinicians push the cardiovascular system hard enough to unmask disease while keeping the patient safe? This question defines the monitoring responsibilities of the EKG technician, who must balance the diagnostic value of achieving target heart rate against the imperative of recognizing when continued exertion threatens patient welfare.

SECTION 2

Core Principles of Stress Test Monitoring

Effective stress test monitoring rests on several foundational principles that every EKG technician must internalize before entering the stress lab. These principles govern what to watch, when to intervene, and how to communicate findings to the supervising physician in real time. Understanding them transforms the technician from a passive recorder of data into an active participant in clinical decision-making.

1

Continuous EKG Surveillance

All 12 leads must be monitored throughout the test. The technician watches for ST-segment depression or elevation, arrhythmias (PVCs, ventricular tachycardia, heart blocks), and rate-related conduction changes. Lead placement must remain secure despite patient movement and perspiration.
2

Hemodynamic Assessment

Blood pressure is measured at baseline, at each protocol stage, at peak exercise, and during recovery. A systolic drop of ≥10 mmHg from baseline despite increasing workload is a critical finding suggesting left ventricular dysfunction or outflow obstruction.
3

Symptom Monitoring

The technician continuously assesses the patient for chest pain, dyspnea, dizziness, claudication, and excessive fatigue. The Borg Rating of Perceived Exertion (RPE) scale is used to quantify subjective effort. Symptoms are documented with their onset time and stage.
4

Target Heart Rate Achievement

A diagnostically adequate test typically requires the patient to reach at least 85% of age-predicted maximum heart rate (220 − age). Failure to achieve this threshold reduces the test's sensitivity for detecting ischemia.
5

Termination Criteria Awareness

The technician must know absolute and relative indications for stopping the test immediately. Absolute criteria include sustained ventricular tachycardia, ST elevation ≥1 mm in leads without Q waves, and a drop in systolic BP below standing resting value with signs of ischemia.
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KEY TAKEAWAY

Think of the EKG technician during a stress test as an air traffic controller. Just as the controller simultaneously monitors radar screens, weather data, and pilot communications to ensure aircraft safety, the technician tracks the EKG waveform, blood pressure readings, patient symptoms, and exercise workload in parallel. Missing any single data stream could mean missing a critical safety event. The skill lies not just in watching each parameter, but in integrating them into a real-time clinical picture.

SECTION 3

Visual Explanation — The Stress Test Monitoring Workflow

STRESS TEST MONITORING WORKFLOWPRE-TESTBaseline EKG, BP,Consent, LeadsEXERCISEGraded stages,Continuous monitorPEAKTarget HR check,Max BP, SymptomsRECOVERYCool-down, monitor6-10 min postContinuous Monitoring Parameters🫀 EKG• 12-lead rhythm• ST-segment analysis• Arrhythmia detection• Rate & axis changes📊 Hemodynamics• Systolic / Diastolic BP• Heart rate trend• Rate-pressure product• HR recovery index👤 Clinical Signs• Chest pain / angina• Dyspnea severity• Borg RPE scale• Skin color, diaphoresisDecision Points During TestABSOLUTE STOP CRITERIA• Sustained VT / VF• ST elevation ≥1 mm (non-Q leads)• SBP drop >10 mmHg + ischemia signs• Moderate-severe angina (≥3/4 scale)• CNS symptoms (ataxia, near-syncope)RELATIVE STOP CRITERIA• Marked ST depression (>2 mm horizontal/downsloping)• Increasing chest pain• Fatigue, dyspnea, leg cramps, claudication• Hypertensive response (SBP >250 or DBP >115)• Arrhythmias other than sustained VT
The stress test monitoring workflow progresses through four phases: pre-test preparation, graded exercise, peak exertion, and recovery. The technician continuously monitors three parameter domains — EKG, hemodynamics, and clinical signs — and must be prepared to invoke absolute or relative termination criteria at any point.

The diagram above illustrates the sequential workflow of a stress test, but it is essential to understand that monitoring is not a linear checklist — it is a continuous, simultaneous process. The EKG technician must visually scan the rhythm strip, verbally check in with the patient regarding symptoms, and manually inflate the blood pressure cuff at prescribed intervals, all while documenting findings in real time. Each phase of the test carries its own set of monitoring priorities: during pre-test, the focus is on establishing clean baseline tracings and confirming the absence of contraindications; during exercise, attention shifts to dynamic EKG changes and progressive hemodynamic response; at peak exertion, the technician confirms whether target heart rate has been achieved and watches for maximal ischemic changes; and during recovery, the concern is that some arrhythmias and ST-segment changes emerge only after exercise cessation.

SECTION 4

Physiological Basis & Key Calculations

Although stress test monitoring is primarily a clinical observation skill, several quantitative parameters guide the technician's assessment and are integral to test interpretation. Understanding the physiological rationale behind these calculations strengthens the technician's ability to anticipate problems and communicate findings effectively. The most fundamental calculation is the age-predicted maximum heart rate (APMHR), which serves as the benchmark for gauging exercise adequacy.

AGE-PREDICTED MAXIMUM HEART RATE

APMHR=220−age(years)APMHR = 220 − age (years)

APMHR is expressed in beats per minute (bpm). The diagnostic threshold for a stress test is typically 85% of APMHR. For example, a 60-year-old patient has an APMHR of 160 bpm and a target of 136 bpm.

RATE-PRESSURE PRODUCT (RPP)

RPP=HeartRate×SystolicBloodPressureRPP = Heart Rate × Systolic Blood Pressure

Also called the double product, RPP estimates myocardial oxygen demand (MVO₂). Normal peak RPP is approximately 25,000–40,000. Values exceeding 40,000 suggest maximal cardiac workload, while angina consistently appearing at a specific RPP threshold (the ischemic threshold) indicates reproducible demand-supply mismatch.

HEART RATE RECOVERY (HRR)

HRR=HRpeak−HR(1minpost−exercise)HRR = HR_peak − HR_(1 min post-exercise)

Heart rate recovery reflects parasympathetic reactivation after exercise cessation. An HRR of less than 12 bpm after one minute of recovery (in an upright cool-down) is an independent predictor of increased mortality and should be flagged by the technician.

METABOLIC EQUIVALENTS (METs)

1MET=3.5mLO2/kg/min1 MET = 3.5 mL O₂ / kg / min

METs quantify the energy cost of exercise relative to resting metabolism. Each stage of the Bruce protocol corresponds to a defined MET level: Stage 1 ≈ 4.6 METs, Stage 2 ≈ 7.0 METs, Stage 3 ≈ 10.1 METs. Achieving fewer than 5 METs is associated with poor prognosis and higher cardiac event rates.

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KEY TAKEAWAY

The rate-pressure product is analogous to the fuel consumption of a car engine: heart rate is the RPM, and systolic blood pressure is the load on the engine. Multiplying them tells you how hard the cardiac "engine" is working. When angina appears at the same RPP regardless of how the workload is achieved, you have identified the patient's ischemic threshold — the redline on their cardiovascular tachometer.

SECTION 5

EKG Changes During Stress Testing — A Detailed Breakdown

The EKG technician must be able to rapidly distinguish normal exercise-induced changes from pathological findings. During progressive exercise, several normal physiological alterations in the EKG occur: heart rate increases, the PR interval shortens, the P-wave amplitude may increase, and the J-point may become depressed due to atrial repolarization overlap. These must not be confused with the pathological ST-segment changes that constitute the hallmark of exercise-induced ischemia. Understanding the morphology, magnitude, and distribution of ST-segment changes is the most important visual skill the technician develops.

ST-SEGMENT MORPHOLOGY: NORMAL vs. ISCHEMIC PATTERNSNORMALUpsloping ST depressionJ-pointST rapidly returns to baselineISCHEMIC — Horizontal≥1 mm at 80 ms past J-point≥1mmFlat ST segment = ischemiaISCHEMIC — DownslopingMost specific for CADDownsloping = high specificityST ELEVATION — INJURY PATTERNAbsolute indication for test termination≥1 mm elevationST elevation in leads without diagnostic Q waves→ STOP TEST IMMEDIATELYMEASUREMENT TECHNIQUE1. Identify the J-point (junction of QRS endand ST-segment onset)2. Measure ST deviation 60-80 ms afterthe J-point relative to the PQ baseline3. Classify morphology: upsloping,horizontal, or downslopingPositive test: ≥1 mm horizontal/downslopingST depression at 60-80 ms past J-point
This diagram contrasts normal upsloping J-point depression (left, green) with the two pathological patterns of ST-segment depression indicating ischemia: horizontal (center, red) and downsloping (right, red). The bottom left panel shows ST elevation, an injury pattern requiring immediate test termination. The measurement technique panel (bottom right) summarizes the systematic approach to ST-segment analysis.
ST MorphologyClinical SignificanceAction Required
Upsloping depression (< 1.5 mm)Normal physiological response; atrial repolarization wave (Ta wave) overlapContinue test; document finding
Horizontal depression ≥ 1 mmSuggestive of subendocardial ischemia; high sensitivity for CADAlert physician; document leads, stage, and symptoms; relative indication to stop if progressive
Downsloping depression ≥ 1 mmHighly specific for significant CAD; often correlates with multi-vessel diseaseAlert physician immediately; strong relative indication to stop
ST elevation ≥ 1 mm (no Q waves)Transmural ischemia or vasospasm; acute injury patternABSOLUTE indication to stop test immediately

ST-segment classification and corresponding actions during stress testing

SECTION 6

Worked Example — Monitoring a Bruce Protocol Stress Test

The following scenario walks through a typical exercise stress test, illustrating how the EKG technician applies monitoring principles in real time. The patient is a 55-year-old male referred for evaluation of atypical chest pain. He has a history of hypertension, controlled with medication, and no known coronary artery disease.

Bruce Protocol Stress Test — Monitoring Walkthrough

  1. 1

    Step 1 — Pre-Test Preparation

    Confirm informed consent is signed. Obtain a supine and standing 12-lead resting EKG. Record baseline vitals: HR 72 bpm, BP 138/82 mmHg. Calculate the age-predicted maximum heart rate: APMHR = 220 − 55 = 165 bpm. Determine the 85% target: 165 × 0.85 = 140 bpm. Verify lead placement is secure and that all 12 leads display clean tracings without artifact. Review the patient's medication list — note that beta-blockers may blunt the heart rate response.
    APMHR = 165 bpm; Target HR = 140 bpm
  2. 2

    Step 2 — Stage 1 (1.7 mph, 10% grade, ~4.6 METs)

    Three minutes into Stage 1, the patient walks at a comfortable pace. HR increases to 92 bpm, BP rises to 148/80 mmHg. The EKG shows sinus rhythm with normal ST segments. No symptoms reported. The technician documents findings and obtains a Stage 1 rhythm strip and blood pressure recording.
    RPP = 92 × 148 = 13,616 — low myocardial demand; normal response
  3. 3

    Step 3 — Stage 2 (2.5 mph, 12% grade, ~7.0 METs)

    HR increases to 118 bpm, BP 162/78 mmHg. The technician notices 0.5 mm of upsloping J-point depression in leads V5–V6. This is within normal physiological limits. The patient reports mild exertion (Borg RPE 11/20, "light"). No chest pain or dyspnea. The technician documents the J-point finding and continues monitoring.
    RPP = 118 × 162 = 19,116 — moderate demand; upsloping ST = normal variant
  4. 4

    Step 4 — Stage 3 (3.4 mph, 14% grade, ~10.1 METs)

    HR reaches 138 bpm, BP 178/76 mmHg. The technician observes 1.5 mm of horizontal ST depression in leads II, III, aVF, and V4–V6. The patient reports mild substernal pressure (angina score 1/4). The technician immediately alerts the supervising physician, documents the onset time, the leads involved, the ST morphology, and the concurrent symptom. The physician decides to continue the test with close observation to reach target heart rate.
    POSITIVE FINDING: 1.5 mm horizontal ST depression + angina at RPP = 24,564
  5. 5

    Step 5 — Test Termination and Recovery

    At 10 minutes of exercise (early Stage 4), HR reaches 142 bpm (exceeds 85% target). The patient's chest pressure increases to 2/4. The physician orders test termination. The technician reduces treadmill speed, then stops the belt. Continuous EKG monitoring and BP recording continue throughout 6 minutes of supine recovery. At 1 minute post-exercise, HR = 122 bpm, giving an HRR = 142 − 122 = 20 bpm (normal, > 12). ST depression resolves by 4 minutes of recovery. All findings are documented.
    HRR = 20 bpm (normal); ST changes resolved in recovery; test positive for ischemia

SECTION 7

Exercise vs. Pharmacological Stress Testing — Monitoring Differences

Not all patients can exercise to diagnostic levels. Patients with significant orthopedic limitations, severe peripheral vascular disease, neurological conditions, or deconditioning may require pharmacological stress testing. The monitoring responsibilities of the EKG technician differ in important ways between exercise and pharmacological protocols, though the foundational principles remain the same. Understanding these differences is essential because the side-effect profiles and timing of ischemic events vary by agent.

ParameterExercise Stress TestPharmacological Stress (Dobutamine)Pharmacological Stress (Adenosine/Regadenoson)
MechanismIncreases myocardial O₂ demand via heart rate and BP elevationSynthetic catecholamine; increases HR and contractility directlyCoronary vasodilator; creates flow heterogeneity ("steal" phenomenon)
EKG monitoring focusST-segment changes, exercise-induced arrhythmiasTachyarrhythmias (VT, AF), ST changes, wall motion (if echo)AV block, bradycardia, ST changes less prominent
Common side effects to monitorFatigue, leg pain, dyspnea, chest painPalpitations, chest pain, headache, tremor, hypotensionFlushing, dyspnea, chest tightness, headache, AV block
Reversal agentN/A — stop treadmillBeta-blocker (esmolol) if neededAminophylline (theophylline IV) for adenosine; caffeine avoidance pre-test
BP monitoring frequencyEvery 2–3 minutes (each stage)Every 1–3 minutes; dose-dependentEvery 1–2 minutes during infusion

Monitoring differences between exercise and pharmacological stress testing modalities

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KEY TAKEAWAY

Whether the stress is achieved through exercise or pharmacology, the technician's core mission is identical: watch the EKG, track the blood pressure, listen to the patient, and know the termination criteria. The difference lies in the specific side effects to anticipate. Dobutamine can trigger dangerous tachyarrhythmias; adenosine can cause high-degree AV block. Knowing which agent is being used tells you which complications to watch for — much like how a pilot adjusts checklists based on whether the aircraft is propeller-driven or jet-powered, even though the principles of safe flight are universal.

SECTION 8

Connection to Advanced Cardiac Diagnostics

Standard exercise EKG stress testing provides valuable diagnostic information, but it has well-known limitations in sensitivity (approximately 68%) and specificity (approximately 77%) for detecting obstructive coronary artery disease. Advanced imaging modalities are frequently combined with stress protocols to improve diagnostic accuracy, and EKG technicians increasingly work alongside nuclear medicine technologists and cardiac sonographers in these integrated procedures. Understanding how basic stress test monitoring connects to these advanced studies enriches the technician's clinical perspective and may expand career opportunities.

FeatureStandard Exercise EKG Stress TestStress EchocardiographyNuclear Myocardial Perfusion Imaging
What it addsEKG changes + symptoms + hemodynamicsReal-time wall motion assessment at rest and stressRadiotracer uptake maps showing perfusion defects
Sensitivity~68%~85%~87%
EKG technician rolePrimary operator and safety monitorEKG monitoring while sonographer images the heartEKG monitoring during stress phase; nuclear tech handles imaging
Key monitoring differenceEKG is the sole diagnostic modalityMust hold patient still immediately post-exercise for echo windowRadiotracer injection timed to peak stress; technician must alert team at target HR

Comparison of standard exercise EKG stress test with advanced imaging-enhanced stress modalities

As healthcare systems adopt more sophisticated cardiac diagnostic protocols, the EKG technician's role continues to evolve. Familiarity with these advanced modalities not only improves the technician's understanding of why stress test monitoring parameters matter, but also prepares them for interdisciplinary team environments where the stress lab may incorporate echocardiography, nuclear imaging, or cardiac MRI. Regardless of the imaging overlay, the technician's fundamental responsibility — continuous, vigilant monitoring of patient safety — remains the irreducible core of the procedure.

SECTION 9

Practice Problems

Question 1: CONCEPTUAL

Explain why a resting 12-lead EKG may appear completely normal in a patient with significant coronary artery disease, and describe how exercise stress testing overcomes this limitation.
Show answer
At rest, even a coronary artery with significant stenosis (e.g., 70% occlusion) may permit sufficient blood flow to meet the myocardium's relatively low metabolic demands. Because there is no supply-demand mismatch at rest, the EKG shows no ischemic changes — the ST segments remain at baseline, and the patient is asymptomatic. During exercise, myocardial oxygen demand rises sharply as heart rate, contractility, and blood pressure increase. The stenotic vessel cannot proportionally increase flow, creating a supply-demand mismatch that manifests as subendocardial ischemia. This ischemia alters ventricular repolarization, producing measurable ST-segment depression on the EKG. Thus, exercise stress testing uncovers latent ischemia by pushing the cardiovascular system beyond the compensatory capacity of diseased coronary arteries.

Question 2: BASIC CALCULATION

A 48-year-old female patient is scheduled for a Bruce protocol stress test. Calculate her age-predicted maximum heart rate, her 85% target heart rate, and the rate-pressure product at the moment her heart rate reaches 130 bpm with a systolic blood pressure of 172 mmHg.
Show answer
The age-predicted maximum heart rate (APMHR) is calculated as 220 − 48 = 172 bpm. The 85% target heart rate is 172 × 0.85 = 146 bpm (rounded to the nearest whole number). At the specified moment, the rate-pressure product (RPP) is HR × SBP = 130 × 172 = 22,360. This RPP indicates a moderate level of myocardial oxygen demand. The patient has not yet reached her target heart rate of 146 bpm, so the test has not yet achieved diagnostic adequacy. The technician should document the RPP and continue monitoring as exercise intensity increases.

Question 3: INTERMEDIATE

During Stage 3 of a Bruce protocol stress test, you observe 2.0 mm of downsloping ST depression in leads V4–V6. The patient's heart rate is 148 bpm, systolic BP is 164 mmHg, and the patient reports no chest pain but mild dyspnea. Is this a positive test? Should you stop the test? What specific information must you communicate to the supervising physician?
Show answer
Yes, this constitutes a positive stress test. The criterion for a positive EKG finding is ≥1 mm of horizontal or downsloping ST-segment depression measured 60–80 ms past the J-point, and 2.0 mm of downsloping depression clearly exceeds this threshold. The downsloping morphology is the most specific pattern for significant coronary artery disease. Regarding test termination, 2.0 mm of ST depression is a relative indication for stopping, not an absolute one, particularly in the absence of chest pain. The technician should immediately alert the physician and communicate the following: the leads involved (V4–V6, suggesting the left anterior descending territory), the magnitude (2.0 mm), the morphology (downsloping), the concurrent heart rate and blood pressure (RPP = 148 × 164 = 24,272), and the patient's symptom status (no angina, mild dyspnea). The decision to continue or stop is the physician's, but the technician's thorough, rapid communication enables an informed decision.

Question 4: APPLIED

You are monitoring a 70-year-old patient undergoing a dobutamine stress echocardiogram. At peak dose, the patient's heart rate is 134 bpm (APMHR = 150, target = 128 — exceeded). Suddenly, you observe a run of 8 consecutive wide-complex beats at a rate of approximately 180 bpm on the EKG monitor, followed by a return to sinus rhythm. The patient states he felt "fluttering" during the episode. Describe your immediate actions and your documentation responsibilities.
Show answer
The 8-beat run of wide-complex tachycardia at 180 bpm is consistent with nonsustained ventricular tachycardia (NSVT), which is a known complication of dobutamine infusion due to its catecholaminergic effects. Immediate actions include: (1) verbally alert the supervising physician and the cardiac sonographer, (2) save/print the rhythm strip capturing the event, (3) confirm the patient is hemodynamically stable by checking blood pressure and level of consciousness, and (4) prepare esmolol (the reversal agent for dobutamine) in case the physician orders its administration. Although NSVT (< 30 seconds) is technically a relative termination criterion, the physician may choose to stop the dobutamine infusion given that target heart rate was already achieved. Documentation must include: the exact time of the arrhythmia, the number of beats (8), the morphology (wide-complex), the approximate rate (180 bpm), the patient's symptoms ("fluttering"), hemodynamic status at the time (HR 134, BP), the dobutamine dose at which it occurred, and the physician's response to the notification. This documentation is critical for both the clinical record and legal protection.

Question 5: CRITICAL THINKING

A patient completes a stress test reaching 95% of his APMHR with no ST-segment changes, no arrhythmias, and no symptoms, yet his heart rate recovery (HRR) at one minute is only 8 bpm. The supervising physician mentions that this is a concerning finding. Analyze why a test that is negative for ischemia can still yield prognostically important information through HRR. What physiological mechanism underlies the prognostic significance of a blunted HRR, and how does this finding change the clinical picture?
Show answer
Heart rate recovery reflects the reactivation of parasympathetic (vagal) tone after exercise cessation, combined with withdrawal of sympathetic drive. An HRR of less than 12 bpm at one minute of recovery is abnormal and has been validated as an independent predictor of all-cause mortality, regardless of the presence or absence of inducible ischemia on the EKG. The physiological basis is that impaired vagal reactivation suggests autonomic dysfunction, which is associated with a pro-arrhythmic state and increased cardiovascular risk. Chronic conditions such as heart failure, diabetes, and metabolic syndrome are associated with reduced vagal tone. In this case, although the stress EKG is negative for ischemia (suggesting that the patient's coronary arteries can meet increased metabolic demand without significant supply-demand mismatch), the blunted HRR of 8 bpm reveals an underlying autonomic abnormality that confers elevated risk. This demonstrates an important principle: stress test interpretation goes beyond binary positive/negative ischemia calls. The technician who understands HRR significance can appropriately flag this finding for the physician, contributing to a more comprehensive risk assessment that may prompt further evaluation such as Holter monitoring, echocardiography for left ventricular function, or metabolic workup.

SUMMARY

Lesson Summary

Summary

Stress test monitoring is a multidimensional patient safety responsibility that requires the EKG technician to simultaneously track continuous 12-lead EKG for ST-segment changes and arrhythmias, hemodynamic parameters including serial blood pressure and heart rate, and clinical symptoms such as angina, dyspnea, and exertional tolerance assessed via the Borg RPE scale. Key quantitative benchmarks include the age-predicted maximum heart rate (220 − age) with an 85% target for diagnostic adequacy, the rate-pressure product estimating myocardial oxygen demand, and heart rate recovery as an independent prognostic marker (abnormal if < 12 bpm at one minute).

The technician must recognize absolute termination criteria — including sustained ventricular tachycardia, ST elevation ≥1 mm in non-Q-wave leads, and systolic BP drop with ischemic signs — versus relative termination criteria such as marked ST depression, escalating symptoms, or hypertensive response. Whether the stress is achieved through exercise or pharmacological agents (dobutamine, adenosine, regadenoson), the technician's core responsibility is vigilant, integrated monitoring that protects the patient while enabling a diagnostically adequate examination.

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